However, the Y563A mutation affected only the gE basolateral cell-surface redistribution function. The Y467A mutation affected the gE endocytosis, gE trans-Golgi network (TGN) recycling, and gE virion incorporation properties. However, they were slightly larger than the gE CT-null virus plaques. The acyl-homoserine lactone (AHL)-mediated quorum-sensing (QS) system dependent on a pair of luxI and luxR homologs, tofI and tofR, is the primary cell-to-cell signaling mechanism determining the virulence of this bacterium.Production of toxoflavin, a major virulence factor of B. The results revealed that disruption of the tyrosine-based motifs, 467YTSL 470 and 563YTVV 566, individually produced smaller plaque phenotypes than the wild type. Burkholderia glumae is the chief causal agent for bacterial panicle blight of rice. In the absence of the directional cues provided by the gravity vector, the plant is especially perceptive to other cues such as light.
Even less is known about plants’ response to partial or reduced gravity levels.
A panel of gE-CT specific mutant viruses was constructed and characterized, in vitro, with respect to their plaque phenotypes, gE recycling and gE basolateral membrane targeting. The response of plants to the spaceflight environment and microgravity is still not well understood, although research has increased in this area. In this study, our goal was to map the gE CT sub-domains that contribute to virus cell-to-cell spread property. Also, the gE CT contributes to virus cell-to-cell spread and anterograde neuronal transport. Bovine herpesvirus envelope glycoprotein E (gE) and, in particular, the gE cytoplasmic tail (CT) is a virulence determinant in cattle.
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